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Anantin

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Competitive antagonist of atrial natriuretic peptide NPR-A receptors. It binds competitively to atrial natriuretic peptide (ANP) receptors from bovine adrenal cortex and inhibits the ANP-induced intracellular cGMP accumulation in bovine aorta smooth muscle cells. Can also reduce the increase in cGMP produced by ANP or BNP (brain natriuretic peptide) in pregnant guinea pig myometrium, but has no effect on relaxation induced by either peptide. Blocks the ANP-induced human sperm acrosome reaction without affecting the acrosomal exocytosis or sperm motility. Also exhibits major and deleterious nonspecific (and even cytotoxic) effects on human fat cells, as it displays noncompetitive antagonism and exerts an inhibitory action on basal and beta-adrenergic receptor-induced lipolytic response.

Below are the list of possible Anantin products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Anantin

 Anantin ELISA Kit
 Anantin Recombinant
 Anantin Antibody
Also known as Anantin (GC-A blocker) (NPR-A antagonist).
Competitive antagonist of atrial natriuretic peptide NPR-A receptors. It binds competitively to atrial natriuretic peptide (ANP) receptors f
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rom bovine adrenal cortex and inhibits the ANP-induced intracellular cGMP accumulation in bovine aorta smooth muscle cells. Can also reduce the increase in cGMP produced by ANP or BNP (brain natriuretic peptide) in pregnant guinea pig myometrium, but has no effect on relaxation induced by either peptide. Blocks the ANP-induced human sperm acrosome reaction without affecting the acrosomal exocytosis or sperm motility. Also exhibits major and deleterious nonspecific (and even cytotoxic) effects on human fat cells, as it displays noncompetitive antagonism and exerts an inhibitory action on basal and beta-adrenergic receptor-induced lipolytic response.
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