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Band 3 anion transport protein

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Below are the list of possible Band 3 anion transport protein products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Band 3 anion transport protein

 Band 3 anion transport protein ELISA Kit
 Band 3 anion transport protein Recombinant
 Band 3 anion transport protein Antibody
Also known as Band 3 anion transport protein (Anion exchange protein 1) (AE 1) (Anion exchanger 1) (MEB3) (Solute carrier family 4 member 1) (CD antigen CD233).
SLC4A1: Band 3 is the major integral glycoprotein of the erythrocyte membrane. Band 3 has two functional domains. Its integral domain mediates a 1:1 exchange of inorganic anions across the membrane, whereas its cytoplasmic domain provid
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es binding sites for cytoskeletal proteins, glycolytic enzymes, and hemoglobin. Defects in SLC4A1 are the cause of elliptocytosis type 4 (EL4). EL4 is a Rhesus-unlinked form of hereditary elliptocytosis, a genetically heterogeneous, autosomal dominant hematologic disorder. It is characterized by variable hemolytic anemia and elliptical or oval red cell shape. Defects in SLC4A1 are the cause of spherocytosis type 4 (SPH4); also known as hereditary spherocytosis type 4 (HS4). Spherocytosis is a hematologic disorder leading to chronic hemolytic anemia and characterized by numerous abnormally shaped erythrocytes which are generally spheroidal. Defects in SLC4A1 are the cause of renal tubular acidosis, distal, autosomal dominant (AD-dRTA). A disease characterized by reduced ability to acidify urine, variable hyperchloremic hypokalemic metabolic acidosis, nephrocalcinosis, and nephrolithiasis. Defects in SLC4A1 are the cause of renal tubular acidosis, distal, with hemolytic anemia (dRTA-HA). A disease characterized by the association of hemolytic anemia with distal renal tubular acidosis, the reduced ability to acidify urine resulting in variable hyperchloremic hypokalemic metabolic acidosis, nephrocalcinosis, and nephrolithiasis. Defects in SLC4A1 are the cause of renal tubular acidosis, distal, with normal red cell morphology (dRTA-NRC). A disease characterized by reduced ability to acidify urine, variable hyperchloremic hypokalemic metabolic acidosis, nephrocalcinosis, and nephrolithiasis. Belongs to the anion exchanger (TC 2.A.31) family.

Protein type: Membrane protein, integral; Membrane protein, multi-pass; Transporter; Transporter, SLC family

Cellular Component: basolateral plasma membrane; cell surface; cortical cytoskeleton; integral to plasma membrane; membrane; plasma membrane; Z disc

Molecular Function: actin binding; anion:anion antiporter activity; ankyrin binding; bicarbonate transmembrane transporter activity; chloride transmembrane transporter activity; enzyme binding; inorganic anion exchanger activity; protein binding; protein C-terminus binding; protein homodimerization activity

Biological Process: anion transport; bicarbonate transport; chloride transport; positive regulation of T cell proliferation; protein oligomerization; regulation of intracellular pH; response to activity; response to arsenic; response to carbon dioxide; response to hydrogen peroxide
 Slc4a1 ELISA Kit
 Slc4a1 Recombinant
 Slc4a1 Antibody
 Ae1 ELISA Kit
 Ae1 Recombinant
 Ae1 Antibody
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