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Bcl-2 homologous antagonist/killer

In the presence of an appropriate stimulus, accelerates programmed cell death by binding to, and antagonizing the anti-apoptotic action of BCL2.

Below are the list of possible Bcl-2 homologous antagonist/killer products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.

Bcl-2 homologous antagonist/killer

 Bcl-2 homologous antagonist/killer ELISA Kit
 Bcl-2 homologous antagonist/killer Recombinant
 Bcl-2 homologous antagonist/killer Antibody
Also known as Bcl-2 homologous antagonist/killer (Apoptosis regulator BAK).
BAK1: In the presence of an appropriate stimulus, accelerates programmed cell death by binding to, and antagonizing the anti- apoptotic action of BCL2 or its adenovirus homolog E1B 19k protein. Low micromolar levels of zinc ions inhibit the promotion of apoptosis. Interacts with BCL2A1. Homodimer. Formation of the homod
imer is zinc-dependent. Forms heterodimers with BCL2, E1B 19k protein, and BCL2L1 isoform Bcl-X(L). Interacts with myxoma virus protein M11L. Expressed in a wide variety of tissues, with highest levels in the heart and skeletal muscle. Belongs to the Bcl-2 family.

Protein type: Apoptosis; Endoplasmic reticulum; Membrane protein, integral; Mitochondrial

Cellular Component: cytosol; endoplasmic reticulum; integral to mitochondrial outer membrane; intracellular; membrane; mitochondrial membrane; mitochondrion; pore complex

Molecular Function: BH domain binding; chaperone binding; heat shock protein binding; identical protein binding; protein binding; protein heterodimerization activity; protein homodimerization activity

Biological Process: apoptosis; B cell apoptosis; B cell homeostasis; B cell negative selection; blood vessel remodeling; caspase activation; caspase activation via cytochrome c; cell proliferation; cytolysis; DNA damage response, signal transduction resulting in induction of apoptosis; establishment and/or maintenance of transmembrane electrochemical gradient; homeostasis of number of cells; leukocyte homeostasis; limb morphogenesis; mitochondrial fusion; myeloid cell homeostasis; negative regulation of cell proliferation; negative regulation of peptidyl-serine phosphorylation; positive regulation of apoptosis; positive regulation of proteolysis; post-embryonic camera-type eye morphogenesis; reduction of endoplasmic reticulum calcium ion concentration; regulation of cell cycle; regulation of mitochondrial membrane permeability; regulation of mitochondrial membrane potential; regulation of protein heterodimerization activity; regulation of protein homodimerization activity; release of cytochrome c from mitochondria; response to fungus; response to gamma radiation; response to mycotoxin; vagina development
 Bak1 ELISA Kit
 Bak1 Recombinant
 Bak1 Antibody
 Bak ELISA Kit
 Bak Recombinant
 Bak Antibody
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