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G protein-activated inward rectifier potassium channel

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This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. This receptor plays a crucial role in regulating the heartbeat.

Below are the list of possible G protein-activated inward rectifier potassium channel products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

G protein-activated inward rectifier potassium channel 1

 G protein-activated inward rectifier potassium channel 1 ELISA Kit
 G protein-activated inward rectifier potassium channel 1 Recombinant
 G protein-activated inward rectifier potassium channel 1 Antibody
Also known as G protein-activated inward rectifier potassium channel 1 (GIRK-1) (Inward rectifier K(+) channel Kir3.1) (KGA) (KGB1) (Potassium channel, inwardly rectifying subfamily J member 3).
GIRK1: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inwa
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rd rectification is mainly due to the blockage of outward current by internal magnesium. This receptor plays a crucial role in regulating the heartbeat. Belongs to the inward rectifier-type potassium channel (TC 1.A.2.1) family. KCNJ3 subfamily.

Protein type: Channel, potassium; Membrane protein, integral; Membrane protein, multi-pass

Cellular Component: cell surface; external side of plasma membrane; integral to plasma membrane; T-tubule

Molecular Function: inward rectifier potassium channel activity; protein binding

Biological Process: potassium ion import; response to electrical stimulus
 Kcnj3 ELISA Kit
 Kcnj3 Recombinant
 Kcnj3 Antibody
 Girk1 ELISA Kit
 Girk1 Recombinant
 Girk1 Antibody
 Kga ELISA Kit
 Kga Recombinant
 Kga Antibody
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G protein-activated inward rectifier potassium channel 2

 G protein-activated inward rectifier potassium channel 2 ELISA Kit
 G protein-activated inward rectifier potassium channel 2 Recombinant
 G protein-activated inward rectifier potassium channel 2 Antibody
Also known as G protein-activated inward rectifier potassium channel 2 (GIRK-2) (BIR1) (Inward rectifier K(+) channel Kir3.2) (KATP-2) (Potassium channel, inwardly rectifying subfamily J member 6).
GIRK2: This potassium channel may be involved in the regulation of insulin secretion by glucose and/or neurotransmitters acting through G-protein-coupled receptors. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as exte
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rnal potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Belongs to the inward rectifier-type potassium channel (TC 1.A.2.1) family. KCNJ6 subfamily.

Protein type: Membrane protein, integral; Membrane protein, multi-pass

Chromosomal Location of Human Ortholog: 21q22.1

Cellular Component: Golgi apparatus; plasma membrane; voltage-gated potassium channel complex

Molecular Function: G-protein activated inward rectifier potassium channel activity; inward rectifier potassium channel activity; protein binding

Biological Process: potassium ion import; potassium ion transport

Disease: Keppen-lubinsky Syndrome
 KCNJ6 ELISA Kit
 KCNJ6 Recombinant
 KCNJ6 Antibody
 GIRK2 ELISA Kit
 GIRK2 Recombinant
 GIRK2 Antibody
 KATP2 ELISA Kit
 KATP2 Recombinant
 KATP2 Antibody
 KCNJ7 ELISA Kit
 KCNJ7 Recombinant
 KCNJ7 Antibody
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G protein-activated inward rectifier potassium channel 3

 G protein-activated inward rectifier potassium channel 3 ELISA Kit
 G protein-activated inward rectifier potassium channel 3 Recombinant
 G protein-activated inward rectifier potassium channel 3 Antibody
Also known as G protein-activated inward rectifier potassium channel 3 (GIRK-3) (Inward rectifier K(+) channel Kir3.3) (Potassium channel, inwardly rectifying subfamily J member 9).
GIRK3: This receptor is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow potassium to flow into the cell rather than out of it. Their voltage dependence
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is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Belongs to the inward rectifier-type potassium channel (TC 1.A.2.1) family. KCNJ9 subfamily.

Protein type: Channel, potassium; Membrane protein, integral; Membrane protein, multi-pass

Chromosomal Location of Human Ortholog: 1q23.2

Cellular Component: integral to plasma membrane; plasma membrane

Molecular Function: G-protein activated inward rectifier potassium channel activity; inward rectifier potassium channel activity; protein binding

Biological Process: potassium ion import
 KCNJ9 ELISA Kit
 KCNJ9 Recombinant
 KCNJ9 Antibody
 GIRK3 ELISA Kit
 GIRK3 Recombinant
 GIRK3 Antibody
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G protein-activated inward rectifier potassium channel 4

 G protein-activated inward rectifier potassium channel 4 ELISA Kit
 G protein-activated inward rectifier potassium channel 4 Recombinant
 G protein-activated inward rectifier potassium channel 4 Antibody
Also known as G protein-activated inward rectifier potassium channel 4 (GIRK-4) (Cardiac inward rectifier) (CIR) (Heart KATP channel) (Inward rectifier K(+) channel Kir3.4) (IRK-4) (KATP-1) (Potassium channel, inwardly rectifying subfamily J member 5).
GIRK4: This potassium channel is controlled by G proteins. Inward rectifier potassium channels are characterized by a greater tendency to allow
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potassium to flow into the cell rather than out of it. Their voltage dependence is regulated by the concentration of extracellular potassium; as external potassium is raised, the voltage range of the channel opening shifts to more positive voltages. The inward rectification is mainly due to the blockage of outward current by internal magnesium. Can be blocked by external barium. Defects in KCNJ5 are the cause of long QT syndrome type 13 (LQT13). It is a heart disorder characterized by a prolonged QT interval on the ECG and polymorphic ventricular arrhythmias. They cause syncope and sudden death in response to excercise or emotional stress, and can present with a sentinel event of sudden cardiac death in infancy. Defects in KCNJ5 are the cause of familial hyperaldosteronism type 3 (FH3). A form of hyperaldosteronism characterized by hypertension secondary to massive adrenal mineralocorticoid production. Like patients with familial hyperaldosteronism type 1 (glucocorticoid-remediable aldosteronism), patients with FH3 present with childhood hypertension, elevated aldosteronism levels, and high levels of the hybrid steroids 18-oxocortisol and 18-hydroxycortisol. However, hypertension and aldosteronism are not reversed by administration of exogenous glucocorticoids and patients require adrenalectomy to control hypertension. Somatic mutations in KCNJ5 have been found in aldosterone-producing adrenal adenomas and can be responsible for aldosteronism associated with cell autonomous proliferation. These are typically solitary, well circumscribed tumors diagnosed between ages 30 and 70. They come to medical attention due to new or worsening hypertension, often with hypokalemia. KCNJ5 mutations produce increased sodium conductance and cell depolarization, which in adrenal glomerulosa cells produces calcium entry, the signal for aldosterone production and cell proliferation. Belongs to the inward rectifier-type potassium channel (TC 1.A.2.1) family. KCNJ5 subfamily.

Protein type: Channel, potassium; Membrane protein, integral; Membrane protein, multi-pass

Chromosomal Location of Human Ortholog: 11q24

Cellular Component: plasma membrane; voltage-gated potassium channel complex

Molecular Function: G-protein activated inward rectifier potassium channel activity; inward rectifier potassium channel activity; protein binding

Biological Process: potassium ion import; potassium ion transport

Disease: Hyperaldosteronism, Familial, Type Iii; Long Qt Syndrome 13
 KCNJ5 ELISA Kit
 KCNJ5 Recombinant
 KCNJ5 Antibody
 GIRK4 ELISA Kit
 GIRK4 Recombinant
 GIRK4 Antibody
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