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Mitofusin

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Essential transmembrane GTPase, which mediates mitochondrial fusion. Fusion of mitochondria occurs in many cell types and constitutes an important step in mitochondrial morphology, which is balanced between fusion and fission, mediated by FZO1 and DNM1, respectively. Functions antagonistically with DNM1. May act by forming membrane contact sites that mediate mitochondrial fusion. Mitochondrial fusion promotes also increased lifespan.

Below are the list of possible Mitofusin products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Mitofusin FZO1

 Mitofusin FZO1 ELISA Kit
 Mitofusin FZO1 Recombinant
 Mitofusin FZO1 Antibody
Also known as Mitofusin FZO1 (Transmembrane GTPase FZO1).
Essential transmembrane GTPase, which mediates mitochondrial fusion. Fusion of mitochondria occurs in many cell types and constitutes an important step in mitochondrial morphology, which is balanced between fusion and fission, mediated by FZO1 and DNM1, respectively. Functions antagonistically with DNM1. May act by forming membrane contact sites that mediate mitochondrial fusion. Mitochondrial fusion promotes also increased lifespan.
 FZO1 ELISA Kit
 FZO1 Recombinant
 FZO1 Antibody
 YBR179C ELISA Kit
 YBR179C Recombinant
 YBR179C Antibody
 YBR1241 ELISA Kit
 YBR1241 Recombinant
 YBR1241 Antibody
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Mitofusin-1

 Mitofusin-1 ELISA Kit
 Mitofusin-1 Recombinant
 Mitofusin-1 Antibody
Also known as Mitofusin-1 (Fzo homolog) (Transmembrane GTPase MFN1).
MFN1: Essential transmembrane GTPase, which mediates mitochondrial fusion. Fusion of mitochondria occurs in many cell types and constitutes an important step in mitochondria morphology, which is balanced between fusion and fission. MFN1 acts independently of the cytoskeleton. Overexpression induces the formation of mitochondri
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al networks. Belongs to the mitofusin family. 3 isoforms of the human protein are produced by alternative splicing.

Protein type: EC 3.6.5.-; Hydrolase; Membrane protein, integral; Membrane protein, multi-pass; Mitochondrial

Chromosomal Location of Human Ortholog: 3q26.33

Cellular Component: mitochondrial outer membrane; mitochondrion

Molecular Function: protein binding

Biological Process: macroautophagy; mitochondrial fusion
 MFN1 ELISA Kit
 MFN1 Recombinant
 MFN1 Antibody
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Mitofusin-2

 Mitofusin-2 ELISA Kit
 Mitofusin-2 Recombinant
 Mitofusin-2 Antibody
Also known as Mitofusin-2 (Hypertension-related protein 1) (Mitochondrial assembly regulatory factor) (HSG protein) (Transmembrane GTPase MFN2).
MFN2: Essential transmembrane GTPase, which mediates mitochondrial fusion. Fusion of mitochondria occurs in many cell types and constitutes an important step in mitochondria morphology, which is balanced between fusion and fission. MFN2 acts independently of the cytoskeleton. It therefore plays a central role in mitochondrial metabolism and may be associated with obesity and/or apoptosis processes. Overexpression induces the formation of mitochondr
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ial networks. Plays an important role in the regulation of vascular smooth muscle cell proliferation. Defects in MFN2 are the cause of Charcot-Marie-Tooth disease type 2A2 (CMT2A2). CMT2A2 is a form of Charcot-Marie-Tooth disease, the most common inherited disorder of the peripheral nervous system. Charcot-Marie-Tooth disease is classified in two main groups on the basis of electrophysiologic properties and histopathology: primary peripheral demyelinating neuropathy or CMT1, and primary peripheral axonal neuropathy or CMT2. Neuropathies of the CMT2 group are characterized by signs of axonal regeneration in the absence of obvious myelin alterations, normal or slightly reduced nerve conduction velocities, and progressive distal muscle weakness and atrophy. Defects in MFN2 are the cause of Charcot-Marie-Tooth disease type 6 (CMT6); also referred to as autosomal dominant hereditary motor and sensory neuropathy VI (HMSN6). CMT6 is an autosomal dominant form of axonal CMT associated with optic atrophy. Belongs to the mitofusin family. 2 isoforms of the human protein are produced by alternative splicing.

Protein type: Cell cycle regulation; Cytoskeletal; EC 3.6.5.-; Hydrolase; Membrane protein, integral; Membrane protein, multi-pass; Mitochondrial

Cellular Component: cytosol; intrinsic to mitochondrial outer membrane; microtubule cytoskeleton; mitochondrial outer membrane; mitochondrion

Molecular Function: GTP binding; GTPase activity; GTPase binding; protein binding; ubiquitin protein ligase binding

Biological Process: blastocyst formation; camera-type eye morphogenesis; mitochondrial fusion; mitochondrial membrane organization and biogenesis; mitochondrion localization; negative regulation of Ras protein signal transduction; protein targeting to mitochondrion
 Mfn2 ELISA Kit
 Mfn2 Recombinant
 Mfn2 Antibody
 Kiaa0214 ELISA Kit
 Kiaa0214 Recombinant
 Kiaa0214 Antibody
 Marf ELISA Kit
 Marf Recombinant
 Marf Antibody
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