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Phospholipase D LiSicTox

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Catalyzes the hydrolysis of sphingomyelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation (By similarity).

Below are the list of possible Phospholipase D LiSicTox products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Phospholipase D LiSicTox-alphaI-1

 Phospholipase D LiSicTox-alphaI-1 ELISA Kit
 Phospholipase D LiSicTox-alphaI-1 Recombinant
 Phospholipase D LiSicTox-alphaI-1 Antibody
Also known as Phospholipase D LiSicTox-alphaI-1 (PLD) (35 kDa dermonecrotic toxin) (Sphingomyelin phosphodiesterase D) (SMD) (SMase D) (Sphingomyelinase D).
Catalyzes the hydrolysis of sphingomyeli
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n. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation ().
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Phospholipase D LiSicTox-alphaIA1a

 Phospholipase D LiSicTox-alphaIA1a ELISA Kit
 Phospholipase D LiSicTox-alphaIA1a Recombinant
 Phospholipase D LiSicTox-alphaIA1a Antibody
Also known as Phospholipase D LiSicTox-alphaIA1a (PLD) (Dermonecrotic toxin 1) (LiRecDT1) (Sphingomyelin phosphodiesterase D 1) (SMD 1) (SMase D 1) (Sphingomyelinase D 1).
Catalyzes the hydrolysis
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of sphingomyelin, lysophosphatidylcholine, and lyso-platelet activating factor but not that of phosphatidylcholine. Shows a high enzymatic activity. Induces dermonecrosis, blood vessel permeability and platelet aggregation (PubMed:9790962). Causes direct nephrotoxicity (PubMed:16005484) and is directly toxic to liver (PubMed:18765244). Also induces hemolysis in a complement-dependent manner (PubMed:9790962) as well as in a complement-independent manner. The hemolysis provoked in a complement-independent manner is composed of several steps. The toxin binds to erythrocyte membranes, hydrolyzes membrane phospholipids (sphingomyelin and lysophosphatidylcholine) thus generating metabolism products that cause hemolysis, probably by provoking an increase of calcium inside cells. The calcium influx is due to the opening of L-type calcium channels, since L-type calcium channel blockers inhibit calcium influx (PubMed:21590705).
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Phospholipase D LiSicTox-alphaIA1bi

 Phospholipase D LiSicTox-alphaIA1bi ELISA Kit
 Phospholipase D LiSicTox-alphaIA1bi Recombinant
 Phospholipase D LiSicTox-alphaIA1bi Antibody
Also known as Phospholipase D LiSicTox-alphaIA1bi (PLD) (Dermonecrotic toxin 1) (LiD1) (LiP1) (P1) (Sphingomyelin phosphodiesterase D 1) (SMD 1) (SMase D 1) (Sphingomyelinase D 1) (recLiD1).
Cataly
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zes the hydrolysis of sphingomyelin with a low activity. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation. Both C5a and the membrane attack complex may play a role in the induction of dermonecrosis. MMP-9 and MMP-2 produced by skin fibroblasts can also contribute to proteolytic tissue destruction.
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Phospholipase D LiSicTox-alphaIA1bii

 Phospholipase D LiSicTox-alphaIA1bii ELISA Kit
 Phospholipase D LiSicTox-alphaIA1bii Recombinant
 Phospholipase D LiSicTox-alphaIA1bii Antibody
Also known as Phospholipase D LiSicTox-alphaIA1bii (PLD) (Dermonecrotic toxin) (Loxtox i4) (Sphingomyelin phosphodiesterase D 1) (SMD 1) (SMase D 1) (Sphingomyelinase D 1).
Catalyzes the hydrolysis
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of sphingomyelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation.
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Phospholipase D LiSicTox-alphaIA2ai

 Phospholipase D LiSicTox-alphaIA2ai ELISA Kit
 Phospholipase D LiSicTox-alphaIA2ai Recombinant
 Phospholipase D LiSicTox-alphaIA2ai Antibody
Also known as Phospholipase D LiSicTox-alphaIA2ai (PLD) (Dermonecrotic toxin) (LiP2) (P2) (Sphingomyelin phosphodiesterase D 2) (SMD 2) (SMase D 2) (Sphingomyelinase D 2).
Catalyzes the hydrolysis
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of sphingomyelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation.
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Phospholipase D LiSicTox-alphaIA2aii

 Phospholipase D LiSicTox-alphaIA2aii ELISA Kit
 Phospholipase D LiSicTox-alphaIA2aii Recombinant
 Phospholipase D LiSicTox-alphaIA2aii Antibody
Also known as Phospholipase D LiSicTox-alphaIA2aii (PLD) (Dermonecrotic toxin) (LiP2) (P2) (LiRecDT2) (Sphingomyelin phosphodiesterase D 2) (SMD 2) (SMase D 2) (Sphingomyelinase D 2).
Catalyzes the
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hydrolysis of sphingomyelin. May also act on other phosphatidyl esters. Shows high hemolytic and catalytic activities. Induces dermonecrosis, blood vessel permeability and platelet aggregation. In addition, also induces hemolysis in a complement-dependent manner (PubMed:9790962) and probably also in a complement-independent manner. The hemolysis provoked in a complement-independent manner may be composed of several steps. The toxin may bind to erythrocyte membranes, may hydrolyze membrane phospholipids (sphingomyelin and lysophosphatidylcholine) thus generating metabolism products that may cause hemolysis, probably by provoking an increase of calcium inside cells. The calcium influx may be due to the opening of L-type calcium channels, since L-type calcium channel blockers inhibit calcium influx.
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Phospholipase D LiSicTox-alphaIA2aiii

 Phospholipase D LiSicTox-alphaIA2aiii ELISA Kit
 Phospholipase D LiSicTox-alphaIA2aiii Recombinant
 Phospholipase D LiSicTox-alphaIA2aiii Antibody
Also known as Phospholipase D LiSicTox-alphaIA2aiii (PLD) (Dermonecrotic toxin) (Loxtox i3) (Sphingomyelin phosphodiesterase D) (SMD) (SMase D) (Sphingomyelinase D).
Catalyzes the hydrolysis of sph
>>>
ingomyelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation ().
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Phospholipase D LiSicTox-alphaIA2bi

 Phospholipase D LiSicTox-alphaIA2bi ELISA Kit
 Phospholipase D LiSicTox-alphaIA2bi Recombinant
 Phospholipase D LiSicTox-alphaIA2bi Antibody
Also known as Phospholipase D LiSicTox-alphaIA2bi (PLD) (Dermonecrotic toxin) (Loxtox i1) (Sphingomyelin phosphodiesterase D) (SMD) (SMase D) (Sphingomyelinase D).
Catalyzes the hydrolysis of sphin
>>>
gomyelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation ().
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Phospholipase D LiSicTox-alphaIA2bii

 Phospholipase D LiSicTox-alphaIA2bii ELISA Kit
 Phospholipase D LiSicTox-alphaIA2bii Recombinant
 Phospholipase D LiSicTox-alphaIA2bii Antibody
Also known as Phospholipase D LiSicTox-alphaIA2bii (PLD) (Dermonecrotic toxin) (Loxtox i2) (Sphingomyelin phosphodiesterase D) (SMD) (SMase D) (Sphingomyelinase D).
Catalyzes the hydrolysis of sphi
>>>
ngomyelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation ().
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Phospholipase D LiSicTox-alphaII1

 Phospholipase D LiSicTox-alphaII1 ELISA Kit
 Phospholipase D LiSicTox-alphaII1 Recombinant
 Phospholipase D LiSicTox-alphaII1 Antibody
Also known as Phospholipase D LiSicTox-alphaII1 (Dermonecrotic toxin 4) (LiRecDT4) (Sphingomyelin phosphodiesterase D 4) (SMD 4) (SMase D 4) (Sphingomyelinase D 4).
Catalyzes the hydrolysis of sphi
>>>
ngomyelin. May also act on other phosphatidyl esters. Shows high hemolytic and catalytic activities. Causes dermonecrosis, induces inflammatory response, platelet aggregation and increases vessel permeability. Shows no lethality when injected at higher dose into mice. May cause complement-dependent hemolysis as well as in a complement-independent manner (PubMed:21590705). The hemolysis provoked in a complement-independent manner may be composed of several steps. The toxin may bind to erythrocyte membranes, may hydrolyze membrane phospholipids (sphingomyelin and lysophosphatidylcholine) thus generating metabolism products that may cause hemolysis, probably by provoking an increase of calcium inside cells. The calcium influx may be due to the opening of L-type calcium channels, since L-type calcium channel blockers inhibit calcium influx.
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Phospholipase D LiSicTox-alphaII2

 Phospholipase D LiSicTox-alphaII2 ELISA Kit
 Phospholipase D LiSicTox-alphaII2 Recombinant
 Phospholipase D LiSicTox-alphaII2 Antibody
Also known as Phospholipase D LiSicTox-alphaII2 (PLD) (Dermonecrotic toxin) (Loxtox i5) (Sphingomyelin phosphodiesterase D) (SMD) (SMase D) (Sphingomyelinase D).
Catalyzes the hydrolysis of sphingo
>>>
myelin. May also act on other phosphatidyl esters. Induces complement-dependent hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation ().
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Phospholipase D LiSicTox-alphaV1

 Phospholipase D LiSicTox-alphaV1 ELISA Kit
 Phospholipase D LiSicTox-alphaV1 Recombinant
 Phospholipase D LiSicTox-alphaV1 Antibody
Also known as Phospholipase D LiSicTox-alphaV1 (PLD) (Dermonecrotic toxin 6) (LiRecDT6) (Sphingomyelin phosphodiesterase D 6) (SMD 6) (SMase D 6) (Sphingomyelinase D 6).
Catalyzes the hydrolysis of
>>>
sphingomyelin. May also act on other phosphatidyl esters. Induces dermonecrosis, blood vessel permeability, edema, and platelet aggregation. Evokes mouse lethality.
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Phospholipase D LiSicTox-betaIA1i

 Phospholipase D LiSicTox-betaIA1i ELISA Kit
 Phospholipase D LiSicTox-betaIA1i Recombinant
 Phospholipase D LiSicTox-betaIA1i Antibody
Also known as Phospholipase D LiSicTox-betaIA1i (PLD) (Dermonecrotic toxin-like I) (LiRecDT3) (Loxtox i6) (P3) (Sphingomyelin phosphodiesterase D 3) (SMD 3) (Smase D 3) (Sphingomyelinase D 3).
Cata
>>>
lyzes the hydrolysis of sphingomyelin with a low activity. May also act on other phosphatidyl esters. Does not cause hemolysis, dermonecrosis, blood vessel blood vessel permeability. Causes swelling and erythema.
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Phospholipase D LiSicTox-betaIA1ii

 Phospholipase D LiSicTox-betaIA1ii ELISA Kit
 Phospholipase D LiSicTox-betaIA1ii Recombinant
 Phospholipase D LiSicTox-betaIA1ii Antibody
Also known as Phospholipase D LiSicTox-betaIA1ii (PLD) (Dermonecrotic toxin-like II) (Loxtox i7) (Sphingomyelin phosphodiesterase D 7) (SMD 7) (Smase D 7) (Sphingomyelinase D 7).
Catalyzes the hydr
>>>
olysis of sphingomyelin with a low activity. May also act on other phosphatidyl esters. Does not cause hemolysis, dermonecrosis, blood vessel permeability and platelet aggregation ().
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Phospholipase D LiSicTox-betaID1

 Phospholipase D LiSicTox-betaID1 ELISA Kit
 Phospholipase D LiSicTox-betaID1 Recombinant
 Phospholipase D LiSicTox-betaID1 Antibody
Also known as Phospholipase D LiSicTox-betaID1 (PLD) (Dermonecrotic toxin 5) (LiRecDT5) (Sphingomyelin phosphodiesterase D 5) (SMD 5) (SMase D 5) (Sphingomyelinase D 5).
Catalyzes the hydrolysis of
>>>
sphingomyelin with a low activity. May also act on other phosphatidyl esters. Causes dermonecrosis, induces inflammatory response, platelet aggregation and increases vessel permeability. Shows no lethality when injected at higher dose into mice. Is less active than LiRecDT4 in phospholipase activity and platelet aggregation. May cause complement-dependent hemolysis. In addition, may also induce hemolysis in a complement-dependent manner and probably also in a complement-independent manner. The hemolysis provoked in a complement-independent manner may be composed of several steps. The toxin may bind to erythrocyte membranes, may hydrolyze membrane phospholipids (sphingomyelin and lysophosphatidylcholine) thus generating metabolism products that may cause hemolysis, probably by provoking an increase of calcium inside cells. The calcium influx may be due to the opening of L-type calcium channels, since L-type calcium channel blockers inhibit calcium influx.
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