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Sodium channel protein

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Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient.

Below are the list of possible Sodium channel protein products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Sodium channel protein

 Sodium channel protein ELISA Kit
 Sodium channel protein Recombinant
 Sodium channel protein Antibody
Also known as Sodium channel protein (Na(+) channel).
Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the volta
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ge difference across the membrane, the protein forms a sodium-selective channel through which Na+ ions may pass in accordance with their electrochemical gradient.
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Sodium channel protein 1 brain

 Sodium channel protein 1 brain ELISA Kit
 Sodium channel protein 1 brain Recombinant
 Sodium channel protein 1 brain Antibody
Also known as Sodium channel protein 1 brain (Sodium channel protein I brain).
Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations
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in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na+ ions may pass in accordance with their electrochemical gradient.
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Sodium channel protein 60E

 Sodium channel protein 60E ELISA Kit
 Sodium channel protein 60E Recombinant
 Sodium channel protein 60E Antibody
Also known as Sodium channel protein 60E (Drosophila ion channel 60) (Drosophila sodium channel 1) (Protein smell-impaired 60E) (Sodium channel 2) (DmNav2).
Mediates the voltage-dependent sodium ion permeability of excitable membranes. Plays a role in processing of olfactory information during the olfactory avoidance response.
 NaCP60E ELISA Kit
 NaCP60E Recombinant
 NaCP60E Antibody
 DIC60 ELISA Kit
 DIC60 Recombinant
 DIC60 Antibody
 DSC1 ELISA Kit
 DSC1 Recombinant
 DSC1 Antibody
 smi60E ELISA Kit
 smi60E Recombinant
 smi60E Antibody
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Sodium channel protein Nach

 Sodium channel protein Nach ELISA Kit
 Sodium channel protein Nach Recombinant
 Sodium channel protein Nach Antibody
Part of a complex that plays a role in tracheal liquid clearance. Probable role in sodium transport ().
 Nach ELISA Kit
 Nach Recombinant
 Nach Antibody
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Sodium channel protein para

 Sodium channel protein para ELISA Kit
 Sodium channel protein para Recombinant
 Sodium channel protein para Antibody
Also known as Sodium channel protein para (Protein paralytic) (Sodium channel 1) (DmNav1).
Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na+ ions may pass in accordance with their electrochemical gradient.
 para ELISA Kit
 para Recombinant
 para Antibody
 CG9907 ELISA Kit
 CG9907 Recombinant
 CG9907 Antibody
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Sodium channel protein type 1 subunit alpha

 Sodium channel protein type 1 subunit alpha ELISA Kit
 Sodium channel protein type 1 subunit alpha Recombinant
 Sodium channel protein type 1 subunit alpha Antibody
Also known as Sodium channel protein type 1 subunit alpha (Sodium channel protein brain I subunit alpha) (Sodium channel protein type I subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.1).
SCN1A: Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the prot
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ein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient. Defects in SCN1A are the cause of generalized epilepsy with febrile seizures plus type 2 (GEFS+2). Generalized epilepsy with febrile seizures-plus refers to a rare autosomal dominant, familial condition with incomplete penetrance and large intrafamilial variability. Patients display febrile seizures persisting sometimes beyond the age of 6 years and/or a variety of afebrile seizure types. GEFS+ is a disease combining febrile seizures, generalized seizures often precipitated by fever at age 6 years or more, and partial seizures, with a variable degree of severity. Defects in SCN1A are a cause of severe myoclonic epilepsy in infancy (SMEI); also called Dravet syndrome. SMEI is a rare disorder characterized by generalized tonic, clonic, and tonic-clonic seizures that are initially induced by fever and begin during the first year of life. Later, patients also manifest other seizure types, including absence, myoclonic, and simple and complex partial seizures. Psychomotor development delay is observed around the second year of life. SMEI is considered to be the most severe phenotype within the spectrum of generalized epilepsies with febrile seizures-plus. Defects in SCN1A are a cause of intractable childhood epilepsy with generalized tonic-clonic seizures (ICEGTC). ICEGTC is a disorder characterized by generalized tonic-clonic seizures beginning usually in infancy and induced by fever. Seizures are associated with subsequent mental decline, as well as ataxia or hypotonia. ICEGTC is similar to SMEI, except for the absence of myoclonic seizures. Defects in SCN1A are the cause of familial hemiplegic migraine type 3 (FHM3). FHM3 is an autosomal dominant severe subtype of migraine with aura characterized by some degree of hemiparesis during the attacks. The episodes are associated with variable features of nausea, vomiting, photophobia, and phonophobia. Age at onset ranges from 6 to 15 years. FHM is occasionally associated with other neurologic symptoms such as cerebellar ataxia or epileptic seizures. A unique eye phenotype of elicited repetitive daily blindness has also been reported to be cosegregating with FHM in a single Swiss family. Defects in SCN1A are the cause of familial febrile convulsions type 3A (FEB3A); also known as familial febrile seizures 3. Febrile convulsions are seizures associated with febrile episodes in childhood without any evidence of intracranial infection or defined pathologic or traumatic cause. It is a common condition, affecting 2-5% of children aged 3 months to 5 years. The majority are simple febrile seizures (generally defined as generalized onset, single seizures with a duration of less than 30 minutes). Complex febrile seizures are characterized by focal onset, duration greater than 30 minutes, and/or more than one seizure in a 24 hour period. The likelihood of developing epilepsy following simple febrile seizures is low. Complex febrile seizures are associated with a moderately increased incidence of epilepsy. Belongs to the sodium channel (TC 1.A.1.10) family. Nav1.1/SCN1A subfamily. 2 isoforms of the human protein are produced by alternative splicing.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Cellular Component: axon; cell soma; membrane; plasma membrane; T-tubule; voltage-gated sodium channel complex; Z disc

Molecular Function: sodium ion binding; voltage-gated sodium channel activity

Biological Process: action potential propagation; adult walking behavior; detection of mechanical stimulus involved in sensory perception of pain; generation of action potential; neuromuscular process controlling posture; regulation of action potential; regulation of membrane potential; sodium ion transport
 Scn1a ELISA Kit
 Scn1a Recombinant
 Scn1a Antibody
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Sodium channel protein type 10 subunit alpha

 Sodium channel protein type 10 subunit alpha ELISA Kit
 Sodium channel protein type 10 subunit alpha Recombinant
 Sodium channel protein type 10 subunit alpha Antibody
Tetrodotoxin-resistant channel that mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which sodium ions may pass in accordance with their electrochemical gradient. Plays a role in neuropathic pain mechanisms.
 Scn10a ELISA Kit
 Scn10a Recombinant
 Scn10a Antibody
 Sns ELISA Kit
 Sns Recombinant
 Sns Antibody
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Sodium channel protein type 11 subunit alpha

 Sodium channel protein type 11 subunit alpha ELISA Kit
 Sodium channel protein type 11 subunit alpha Recombinant
 Sodium channel protein type 11 subunit alpha Antibody
Also known as Sodium channel protein type 11 subunit alpha (NaN) (Sensory neuron sodium channel 2) (Sodium channel protein type XI subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.9).
SCN11A: This protein mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which sodium ions may pass in accordance with their electrochemical gradient. It is a tetrodotoxin-resistant sodium channel isoform. Also involved
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, with the contribution of the receptor tyrosine kinase NTRK2, in rapid BDNF-evoked neuronal depolarization. Belongs to the sodium channel (TC 1.A.1.10) family. Nav1.9/SCN11A subfamily. 3 isoforms of the human protein are produced by alternative splicing.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Cellular Component: axon; plasma membrane

Molecular Function: protein binding; voltage-gated sodium channel activity

Biological Process: generation of action potential; regulation of sensory perception of pain; sodium ion transport
 Scn11a ELISA Kit
 Scn11a Recombinant
 Scn11a Antibody
 Nan ELISA Kit
 Nan Recombinant
 Nan Antibody
 Nat ELISA Kit
 Nat Recombinant
 Nat Antibody
 Sns2 ELISA Kit
 Sns2 Recombinant
 Sns2 Antibody
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Sodium channel protein type 2 subunit alpha

 Sodium channel protein type 2 subunit alpha ELISA Kit
 Sodium channel protein type 2 subunit alpha Recombinant
 Sodium channel protein type 2 subunit alpha Antibody
Also known as Sodium channel protein type 2 subunit alpha (HBSC II) (Sodium channel protein brain II subunit alpha) (Sodium channel protein type II subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.2).
SCN2A: Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient. Defects in SCN2A are the cause of seizures, benign familial infant
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ile type 3 (BFIS3). An autosomal dominant disorder in which afebrile seizures occur in clusters during the first year of life, without neurologic sequelae. Defects in SCN2A are the cause of epileptic encephalopathy early infantile type 11 (EIEE11). EIEE11 is an autosomal dominant seizure disorder characterized by infantile onset of refractory seizures with resultant delayed neurologic development and persistent neurologic abnormalities. Belongs to the sodium channel (TC 1.A.1.10) family. Nav1.2/SCN2A subfamily. 2 isoforms of the human protein are produced by alternative splicing.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Chromosomal Location of Human Ortholog: 2q24.3

Cellular Component: axon; integral to plasma membrane; intrinsic to plasma membrane

Molecular Function: voltage-gated sodium channel activity

Biological Process: generation of action potential; myelination; sodium ion transport

Disease: Epileptic Encephalopathy, Early Infantile, 11; Seizures, Benign Familial Infantile, 3
 SCN2A ELISA Kit
 SCN2A Recombinant
 SCN2A Antibody
 NAC2 ELISA Kit
 NAC2 Recombinant
 NAC2 Antibody
 SCN2A1 ELISA Kit
 SCN2A1 Recombinant
 SCN2A1 Antibody
 SCN2A2 ELISA Kit
 SCN2A2 Recombinant
 SCN2A2 Antibody
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Sodium channel protein type 3 subunit alpha

 Sodium channel protein type 3 subunit alpha ELISA Kit
 Sodium channel protein type 3 subunit alpha Recombinant
 Sodium channel protein type 3 subunit alpha Antibody
Also known as Sodium channel protein type 3 subunit alpha (Sodium channel protein brain III subunit alpha) (Sodium channel protein type III subunit alpha) (Voltage-gated sodium channel subtype III) (Voltage-gated sodium channel subunit alpha Nav1.3).
SCN3A: Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the v
>>>
oltage difference across the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Cellular Component: axon; cell soma; cytoplasm; membrane; voltage-gated sodium channel complex

Molecular Function: calmodulin binding; protein binding; sodium ion binding; voltage-gated sodium channel activity

Biological Process: generation of action potential; nervous system development; response to pyrethroid; response to wounding; sensory perception of pain
 Scn3a ELISA Kit
 Scn3a Recombinant
 Scn3a Antibody
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Sodium channel protein type 4 subunit alpha

 Sodium channel protein type 4 subunit alpha ELISA Kit
 Sodium channel protein type 4 subunit alpha Recombinant
 Sodium channel protein type 4 subunit alpha Antibody
Also known as Sodium channel protein type 4 subunit alpha (Mu-1) (SkM1) (Sodium channel protein skeletal muscle subunit alpha) (Sodium channel protein type IV subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.4).
SCN4A: This protein mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage diff
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erence across the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient. This sodium channel may be present in both denervated and innervated skeletal muscle. Defects in SCN4A are the cause of paramyotonia congenita of von Eulenburg (PMC). PMC is an autosomal dominant channelopathy characterized by myotonia, increased by exposure to cold, intermittent flaccid paresis, not necessarily dependent on cold or myotonia, lability of serum potassium, nonprogressive nature and lack of atrophy or hypertrophy of muscles. In some patients, myotonia is not increased by cold exposure (paramyotonia without cold paralysis). Patients may have a combination phenotype of PMC and HYPP. Defects in SCN4A are a cause of periodic paralysis hypokalemic type 2 (HOKPP2). It is an autosomal dominant disorder manifested by episodic flaccid generalized muscle weakness associated with falls of serum potassium levels. Defects in SCN4A are the cause of periodic paralysis hyperkalemic (HYPP). HYPP is an autosomal dominant channelopathy characterized by episodic flaccid generalized muscle weakness associated with high levels of serum potassium. Concurrence of myotonia is found in HYPP patients. Defects in SCN4A are the cause of periodic paralysis normokalemic (NKPP). NKPP is a disorder closely related to hyperkalemic periodic paralysis, but marked by a lack of alterations in potassium levels during attacks of muscle weakness. Defects in SCN4A are the cause of myotonia SCN4A-related (MYOSCN4A). Myotonia is characterized by sustained muscle tensing that prevents muscles from relaxing normally. Myotonia causes muscle stiffness that can interfere with movement. In some people the stiffness is very mild, while in other cases it may be severe enough to interfere with walking, running, and other activities of daily life. MYOSCN4A is a phenotypically highly variable myotonia aggravated by potassium loading, and often by cold. MYOSCN4A includes myotonia permanens and myotonia fluctuans. In myotonia permanens, the myotonia is generalized and there is a hypertrophy of the muscle, particularly in the neck and the shoulder. Attacks of severe muscle stiffness of the thoracic muscles may be life threatening due to impaired ventilation. In myotonia fluctuans, the muscle stiffness may fluctuate from day to day, provoked by exercise. Defects in SCN4A are the cause of a congenital myasthenic syndrome acetazolamide-responsive (CMSAR). A congenital myasthenic syndrome associated with fatigable generalized weakness and recurrent attacks of respiratory and bulbar paralysis since birth. The fatigable weakness involves lid- elevator, external ocular, facial, limb and truncal muscles and an decremental response of the compound muscle action potential on repetitive stimulation. Belongs to the sodium channel (TC 1.A.1.10) family. Nav1.4/SCN4A subfamily.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass
 Scn4a ELISA Kit
 Scn4a Recombinant
 Scn4a Antibody
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Sodium channel protein type 4 subunit alpha A

 Sodium channel protein type 4 subunit alpha A ELISA Kit
 Sodium channel protein type 4 subunit alpha A Recombinant
 Sodium channel protein type 4 subunit alpha A Antibody
Also known as Sodium channel protein type 4 subunit alpha A (Voltage-gated sodium channel subunit alpha Nav1.4a).
This protein mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na+ ions may pass in accordance w
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ith their electrochemical gradient. This sodium channel may be present in both denervated and innervated skeletal muscle.
 scn4aa ELISA Kit
 scn4aa Recombinant
 scn4aa Antibody
 nav1.4a ELISA Kit
 nav1.4a Recombinant
 nav1.4a Antibody
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Sodium channel protein type 4 subunit alpha B

 Sodium channel protein type 4 subunit alpha B ELISA Kit
 Sodium channel protein type 4 subunit alpha B Recombinant
 Sodium channel protein type 4 subunit alpha B Antibody
Also known as Sodium channel protein type 4 subunit alpha B (Voltage-gated sodium channel subunit alpha Nav1.4b).
This protein mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na+ ions may pass in accordance w
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ith their electrochemical gradient. This sodium channel may be present in both denervated and innervated skeletal muscle.
 scn4ab ELISA Kit
 scn4ab Recombinant
 scn4ab Antibody
 nav1.4b ELISA Kit
 nav1.4b Recombinant
 nav1.4b Antibody
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Sodium channel protein type 5 subunit alpha

 Sodium channel protein type 5 subunit alpha ELISA Kit
 Sodium channel protein type 5 subunit alpha Recombinant
 Sodium channel protein type 5 subunit alpha Antibody
Also known as Sodium channel protein type 5 subunit alpha (HH1) (Sodium channel protein cardiac muscle subunit alpha) (Sodium channel protein type V subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.5).
SCN5A: an integral membrane protein and a voltage-gated sodium channel subunit. Mediates the voltage-dependent sodium ion permeability of excitable membranes. This protein is found
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primarily in cardiac muscle and is responsible for the initial upstroke of the action potential in an electrocardiogram. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient. It is a tetrodotoxin-resistant Na(+) channel isoform. Intracellular calcium levels regulate channel inactivation. Interacts with the PDZ domain of the syntrophin SNTA1, SNTB1 and SNTB2. Interacts with NEDD4, NEDD4L, WWP2 and GPD1L. Defects in this protein are a cause of long QT syndrome type 3 (LQT3). Belongs to the sodium channel (TC 1.A.1.10) family, Nav1.5/SCN5A subfamily. 6 isoforms of the human protein are produced by alternative splicing.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Chromosomal Location of Human Ortholog: 3p21

Cellular Component: caveola; cell surface; endoplasmic reticulum; integral to membrane; intracellular; lateral plasma membrane; plasma membrane; sarcolemma; T-tubule; voltage-gated sodium channel complex; Z disc

Molecular Function: ankyrin binding; calmodulin binding; enzyme binding; fibroblast growth factor binding; nitric-oxide synthase binding; protein binding; protein domain specific binding; protein kinase binding; sodium channel regulator activity; ubiquitin protein ligase binding; voltage-gated sodium channel activity

Biological Process: cardiac muscle contraction; cerebellum development; generation of action potential; membrane depolarization; odontogenesis of dentine-containing teeth; positive regulation of action potential; positive regulation of epithelial cell proliferation; regulation of heart rate; response to denervation involved in regulation of muscle adaptation; sodium ion transport; telencephalon development

Disease: Atrial Fibrillation, Familial, 10; Brugada Syndrome 1; Cardiomyopathy, Dilated, 1e; Long Qt Syndrome 3; Progressive Familial Heart Block, Type Ia; Sick Sinus Syndrome 1, Autosomal Recessive; Sudden Infant Death Syndrome; Ventricular Fibrillation During Myocardial Infarction, Susceptibility To
 SCN5A ELISA Kit
 SCN5A Recombinant
 SCN5A Antibody
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Sodium channel protein type 7 subunit alpha

 Sodium channel protein type 7 subunit alpha ELISA Kit
 Sodium channel protein type 7 subunit alpha Recombinant
 Sodium channel protein type 7 subunit alpha Antibody
Also known as Sodium channel protein type 7 subunit alpha (Putative voltage-gated sodium channel subunit alpha Nax) (Sodium channel protein cardiac and skeletal muscle subunit alpha) (Sodium channel protein type VII subunit alpha).
SCN7A: Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference a
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cross the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient. Belongs to the sodium channel (TC 1.A.1.10) family. SCN7A subfamily.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Chromosomal Location of Human Ortholog: 2q21-q23

Cellular Component: plasma membrane

Biological Process: generation of action potential; sodium ion transport
 SCN7A ELISA Kit
 SCN7A Recombinant
 SCN7A Antibody
 SCN6A ELISA Kit
 SCN6A Recombinant
 SCN6A Antibody
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Sodium channel protein type 8 subunit alpha

 Sodium channel protein type 8 subunit alpha ELISA Kit
 Sodium channel protein type 8 subunit alpha Recombinant
 Sodium channel protein type 8 subunit alpha Antibody
Also known as Sodium channel protein type 8 subunit alpha (Peripheral nerve protein type 4) (PN4) (Sodium channel 6) (NaCh6) (Sodium channel protein type VIII subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.6).
SCN8A: Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across
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the membrane, the protein forms a sodium-selective channel through which Na(+) ions may pass in accordance with their electrochemical gradient. In macrophages and melanoma cells, isoform 5 may participate in the control of podosome and invadopodia formation. Defects in SCN8A are the cause of cognitive impairment with or without cerebellar ataxia (CIAT). A disorder characterized by markedly delayed cognitive and motor development, attention deficit disorder, and cerebellar ataxia. Features include bilateral esophoria, strabismatic amblyopia, unsustained gaze evoked nystagmus on horizontal gaze, ataxic gait, dysmetria in the upper limbs and dysarthria, with normal strength, tone, and reflexes. Defects in SCN8A are the cause of epileptic encephalopathy, early infantile, type 13 (EIEE13). A form of epilepsy characterized by frequent tonic seizures or spasms beginning in infancy with a specific EEG finding of suppression-burst patterns, characterized by high-voltage bursts alternating with almost flat suppression phases. Patients may progress to West syndrome, which is characterized by tonic spasms with clustering, arrest of psychomotor development, and hypsarrhythmia on EEG. EIEE13 is a severe form consisting of early-onset seizures, features of autism, intellectual disability, ataxia, and sudden unexplained death in epilepsy. Belongs to the sodium channel (TC 1.A.1.10) family. Nav1.6/SCN8A subfamily. 5 isoforms of the human protein are produced by alternative splicing.

Protein type: Channel, sodium; Membrane protein, integral; Membrane protein, multi-pass

Cellular Component: cell soma; dendrite; integral to membrane; membrane; plasma membrane; voltage-gated sodium channel complex; Z disc

Molecular Function: protein binding; sodium ion binding; voltage-gated sodium channel activity

Biological Process: adult locomotory behavior; adult walking behavior; generation of action potential; locomotory behavior; muscle development; myelination; neuromuscular process; peripheral nervous system development; response to toxin; sensory perception of sound; sodium ion transport
 Scn8a ELISA Kit
 Scn8a Recombinant
 Scn8a Antibody
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Sodium channel protein type 9 subunit alpha

 Sodium channel protein type 9 subunit alpha ELISA Kit
 Sodium channel protein type 9 subunit alpha Recombinant
 Sodium channel protein type 9 subunit alpha Antibody
Also known as Sodium channel protein type 9 subunit alpha (Nas) (Schwann cell sodium channel) (Sodium channel protein type IX subunit alpha) (Voltage-gated sodium channel subunit alpha Nav1.7).
Mediates the voltage-dependent sodium ion permeability of excitable membranes. Assuming opened or closed conformations in response to the voltage difference across the membrane, the protein forms a sodiu
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m-selective channel through which Na+ ions may pass in accordance with their electrochemical gradient. It is a tetrodotoxin-sensitive Na+ channel isoform. Plays a role in pain mechanisms, especially in the development of inflammatory pain.
 SCN9A ELISA Kit
 SCN9A Recombinant
 SCN9A Antibody
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Proteins Root Name Listing
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