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Twist-related protein

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Probable transcription factor, which may be responsible for the formation of myoepithelial cells in early muscle development in larva and the formation of non-muscle tissues in later bud stages and mesoderm-like structures in the medusa.

Below are the list of possible Twist-related protein products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Twist-related protein

 Twist-related protein ELISA Kit
 Twist-related protein Recombinant
 Twist-related protein Antibody
Probable transcription factor, which may be responsible for the formation of myoepithelial cells in early muscle development in larva and the formation of non-muscle tissues in later bud stages and mesoderm-like structures in the medusa.
 TWIST ELISA Kit
 TWIST Recombinant
 TWIST Antibody
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Twist-related protein 1

 Twist-related protein 1 ELISA Kit
 Twist-related protein 1 Recombinant
 Twist-related protein 1 Antibody
 TWIST1 ELISA Kit
 TWIST1 Recombinant
 TWIST1 Antibody
 TWIST ELISA Kit
 TWIST Recombinant
 TWIST Antibody
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Twist-related protein 2

 Twist-related protein 2 ELISA Kit
 Twist-related protein 2 Recombinant
 Twist-related protein 2 Antibody
TWIST2: Binds to the E-box consensus sequence 5'-CANNTG-3' as a heterodimer and inhibits transcriptional activation by MYOD1, MYOG, MEF2A and MEF2C. Also represses expression of proinflammatory cytokines such as TNFA and IL1B. Involved in postnatal glycogen storage and energy metabolism. Inhibits the premature or ectopic differentiation of preosteoblast cells during osteogenesis, possibly by chang
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ing the internal signal transduction response of osteoblasts to external growth factors. Defects in TWIST2 are the cause of Setleis syndrome (SETLEISS). A focal facial dermal dysplasia characterized by distinctive bitemporal scar-like depressions resembling forceps marks, and additional facial features, including a coarse and leonine appearance, absent eyelashes on both lids or multiple rows on the upper lids, absent Meibomian glands, slanted eyebrows, chin clefting, and hypo- or hyperpigmentation of the skin. Histologically, the bitemporal lesion is an ectodermal dysplasia with near absence of subcutaneous fat, suggesting insufficient migration of neural crest cells into the frontonasal process and the first branchial arch.

Cellular Component: cytoplasm; nucleus; transcription factor complex

Molecular Function: chromatin binding; protein domain specific binding; transcription factor activity

Biological Process: embryonic cranial skeleton morphogenesis; negative regulation of apoptosis; negative regulation of cell proliferation; negative regulation of DNA binding; negative regulation of myeloid cell differentiation; negative regulation of osteoblast differentiation; negative regulation of transcription from RNA polymerase II promoter; negative regulation of transcription, DNA-dependent; negative regulation of tumor necrosis factor production; osteoblast differentiation; regulation of transcription from RNA polymerase II promoter
 Twist2 ELISA Kit
 Twist2 Recombinant
 Twist2 Antibody
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