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Zinc metalloproteinase-disintegrin-like berythractivase

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Potent activator of prothrombin (F2). Does not elicit any hemorrhagic response. Barely inhibits collagen-induced platelet aggregation. Binds neither collagen, nor the jararhagin monoclonal antibody MAJar3. Hydrolyzes the Aalpha-chain of fibrin and fibrinogen, without affecting the Bbeta- and gamma-chains. Is capable of triggering endothelial proinflammatory and procoagulant cell responses, but fails to trigger apoptosis. Induces von Willebrand factor release, and the expression of both ICAM1 and E-selectin (SELE) (without increase in VCAM1) in endothelial cells (HUVEC). Is also able to up-regulate the synthesis of the coagulation factor TF (F3). Enhances nitric oxide (NO) generation, prostacyclin production and interleukin-8 release.

Below are the list of possible Zinc metalloproteinase-disintegrin-like berythractivase products. If you cannot find the target and/or product is not available in our catalog, please click here to contact us and request the product or submit your request for custom elisa kit production, custom recombinant protein production or custom antibody production. Custom ELISA Kits, Recombinant Proteins and Antibodies can be designed, manufactured and produced according to the researcher's specifications.
 

Zinc metalloproteinase-disintegrin-like berythractivase

 Zinc metalloproteinase-disintegrin-like berythractivase ELISA Kit
 Zinc metalloproteinase-disintegrin-like berythractivase Recombinant
 Zinc metalloproteinase-disintegrin-like berythractivase Antibody
Also known as Zinc metalloproteinase-disintegrin-like berythractivase (Snake venom metalloproteinase) (SVMP) (ery1).
Potent activator of prothrombin (F2). Does not elicit any hemorrhagic response.
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Barely inhibits collagen-induced platelet aggregation. Binds neither collagen, nor the jararhagin monoclonal antibody MAJar3. Hydrolyzes the Aalpha-chain of fibrin and fibrinogen, without affecting the Bbeta- and gamma-chains. Is capable of triggering endothelial proinflammatory and procoagulant cell responses, but fails to trigger apoptosis. Induces von Willebrand factor release, and the expression of both ICAM1 and E-selectin (SELE) (without increase in VCAM1) in endothelial cells (HUVEC). Is also able to up-regulate the synthesis of the coagulation factor TF (F3). Enhances nitric oxide (NO) generation, prostacyclin production and interleukin-8 release.
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